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Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vitro model of preeclamptic vascular endothelium: a possible therapeutic role of nicotinic acetylcholine receptor (nAChR) agonists for preeclampsia.
Mimura, Kazuya; Tomimatsu, Takuji; Sharentuya, Namuxila et al.
2010 • In American Journal of Obstetrics and Gynecology, 202 (5), p. 464.e1-6

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Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vivo model of preeclamptic vascular endothelium.pdf

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