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See detailLes dysphasies : de l'évaluation à la rééducation
Maillart, Christelle ULiege; Schelstraete, Marie-Anne

Book published by Elsevier-Masson (2012)

L'étude des troubles du langage oral chez l’enfant a connu un développement considérable à partir des années 1970 avec l’essor des recherches en psychologie du langage, en linguistique, et plus ... [more ▼]

L'étude des troubles du langage oral chez l’enfant a connu un développement considérable à partir des années 1970 avec l’essor des recherches en psychologie du langage, en linguistique, et plus récemment dans le domaine des neurosciences et de la génétique. La prise en charge des troubles langagiers fait également l’objet actuellement d’une attention particulière dans la recherche scientifique. L’objectif principal de ce livre est de rendre accessibles les résultats des recherches récentes sur les troubles dits « dysphasique » aux professionnels concernés par le suivi des enfants présentant cette pathologie. La dysphasie est définie comme un trouble développemental, qui concerne l’élaboration du langage oral, entraînant des difficultés importantes en compréhension et/ou en expression du langage parlé. C’est un trouble spécifique, sévère et persistant qui interfère d’emblée avec la dynamique développementale de l’enfant. La première partie de l’ouvrage expose les approches théo- riques de la dysphasie, actuellement débattues dans la littéra- ture scientifique, et dont la synthèse constitue une aide à l’établissement d’un diagnostic précis ou à la construction de programmes de prévention. La seconde partie présente différents types de rééducation, avec des exemples cliniques, et notamment le courant méthodologique EBP (Evidence-Based Practice), littéralement «pratiques basées sur les preuves», qui vise à aider le clinicien à vérifier la pertinence de ses choix. Illustré par des exemples concrets d’interprétation et d’évaluation des troubles, des stratégies d’élaboration du diagnostic, et comportant les recommandations issues de la littérature internationale, cet ouvrage s’adresse à tous les professionnels concernés par les troubles du langage oral et leur prise en charge : orthophonistes, neuropsychologues, médecins rééducateurs. [less ▲]

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See detailLes dysphasies : nouvelles conceptions théoriques et implications cliniques
Maillart, Christelle ULiege

Conference (2016, February 20)

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See detailLes dysphasies : sémiologie et hypothèses explicatives
Maillart, Christelle ULiege

Conference given outside the academic context (2012)

Detailed reference viewed: 166 (19 ULiège)
See detailLa dysphonie, diagnostic et traitements
FINCK, Camille ULiege

Conference (2007, September)

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See detailLes dysphonies dysfonctionnelles
Morsomme, Dominique ULiege

Conference given outside the academic context (2000)

Detailed reference viewed: 16 (2 ULiège)
See detailDysphonies: évaluation fonctionnelle, diagnostic et traitements
FINCK, Camille ULiege

Scientific conference (2011, February)

Detailed reference viewed: 40 (5 ULiège)
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See detailLa dysplasie fibromusculaire
VAN DAMME, Hendrik ULiege; Quaniers, Janine ULiege; Limet, Raymond ULiege

in Revue Médicale de Liège (1999), 54(12), 935-42

Fibromuscular dysplasia is a rare non-atherosclerotic, non-inflammatory arterial disease. It concerns less than 1% of all occlusive artery lesions, but is more common in young female patients, with a ... [more ▼]

Fibromuscular dysplasia is a rare non-atherosclerotic, non-inflammatory arterial disease. It concerns less than 1% of all occlusive artery lesions, but is more common in young female patients, with a prevalence of 3 to 5% of the arterial lesions in that age group. It mainly attains renal and carotid arteries. The authors discuss the etiopathogeny, the prevalence and treatment of fibromuscular dysplasia. Their own surgical experience with 21 renal and 10 carotid lesions of fibromuscular is exposed. [less ▲]

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See detailDyspnea and stress testing
PIERARD, Luc ULiege; Lancellotti, Patrizio ULiege

in New England Journal of Medicine (2006), 354(8), 871-872

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See detailDyspnee paroxystique chez le sujet sain: le pneumothorax spontane
Duysinx, Bernard ULiege; Nguyen, Delphine; Corhay, Jean-Louis ULiege et al

in Revue Médicale de Liège (2004), 59(4), 205-8

In a healthy individual, a dyspnoea of abrupt onset raises the suspicion of a spontaneous pneumothorax. The clinical examination and standard chest X-ray usually permit the correct diagnosis. The risk of ... [more ▼]

In a healthy individual, a dyspnoea of abrupt onset raises the suspicion of a spontaneous pneumothorax. The clinical examination and standard chest X-ray usually permit the correct diagnosis. The risk of reccurence can be assessed by the clinical history and examination. The functional consequences (which can be lethal) and the risk of recurrence will guide the short- and long-term therapy. [less ▲]

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See detailDysraphies spinales
Martin, Didier ULiege

Conference (2005, February 25)

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See detailDysregulated circulating miRNAs in pre-eclampsia
Tebache, Linda; Munaut, Carine ULiege; Blacher, Silvia ULiege et al

Conference (2016, September 25)

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See detailDysregulated circulating miRNAs in preeclampsia.
Munaut, Carine ULiege; Tebache, Linda; Blacher, Silvia ULiege et al

in Biomedical Reports (2016), 5(6), 686-692

Preeclampsia (PE) is a pregnancy-related disease with potentially severe consequences with respect to foeto-maternal morbidity and mortality. However, the molecular pathogenesis of PE remains largely ... [more ▼]

Preeclampsia (PE) is a pregnancy-related disease with potentially severe consequences with respect to foeto-maternal morbidity and mortality. However, the molecular pathogenesis of PE remains largely unknown. Recent reports have shown that microRNAs (miRNAs or miRs) may play important roles in the development of PE. Analysing the miRNAs in sera from preeclamptic women may improve our understanding of the pathophysiological mechanisms of the disease. The aim of this retrospective study was to identify whether circulating miRNAs were differentially expressed in PE patients compared with controls. Serum samples from 23 women who developed PE were compared with samples from 44 pregnant controls. Seventeen circulating miRNAs previously described in PE were chosen for evaluation of their expression by reverse transcription quantitative polymerase chain reaction (RT-qPCR). In the maternal serum, the miR-210-3p, miR-210-5p, miR-1233-3p, and miR-574-5p levels were found to be significantly higher in the PE patients than in the controls (P<0.05). Using a logistic regression model, we evaluated the discriminant power of those differentially expressed miRNAs, and the combination of miR-210-5p and miR-574-5p yielded an area under the curve of 0.7223 for discriminating PE patients from the controls. In conclusion, the fact that four circulating miRNAs (miR-210-3p, miR-210-5p, miR-1233-3p, and miR-574-5p) were differentially expressed in the sera of women who developed PE compared with controls confirms the possible pathophysiological role of miRNAs in PE. [less ▲]

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See detailDysregulation of anti-angiogenic agents (sFlt-1, PLGF, and sEndoglin) in preeclampsia--a step forward but not the definitive answer
Foidart, Jean-Michel ULiege; Schaaps, Jean-Pierre ULiege; Chantraine, Frédéric ULiege et al

in Journal of Reproductive Immunology (2009), 82(2), 106-11

Preeclampsia (PE) is a pregnancy-specific syndrome characterized by hypertension, proteinuria and edema, which resolves on placental delivery. It is thought to be the consequence of impaired placentation ... [more ▼]

Preeclampsia (PE) is a pregnancy-specific syndrome characterized by hypertension, proteinuria and edema, which resolves on placental delivery. It is thought to be the consequence of impaired placentation due to inadequate trophoblastic invasion of the maternal spiral arteries. In PE the maternal plasma concentration of free vascular endothelial growth factor (VEGF) and placental growth factor (PlGF) is decreased whereas the concentration of soluble fms-like tyrosine kinase-1 (sFlt-1) and of soluble endoglin (sEng) is increased. These soluble receptors may bind VEGF, PLGF and TGFβ1 and TGFβ3 in the maternal circulation, causing endothelial dysfunction in many maternal tissues. Hence there is a view that the pathogenesis is more or less clarified. According to the vascular theory, poor placentation leads to poor uteroplacental perfusion and hypoxia, which stimulates sFlt-1 and sEng production causing the maternal syndrome. This assumption has been recently challenged. The role of hypoxia as the main stimulus for release of sFlt-1 has been questioned and the role of inflammatory mechanisms has been emphasized. According to this inflammatory theory, poor placentation may predispose more to placental oxidative stress than hypoxia and endothelial dysfunction may be part of a broader disorder of systemic inflammation. Finally, the recent demonstration of activating auto-antibodies to the angiotensin 1 receptor that experimentally play a major pathogenic role in PE further suggests a pleiotropism of aetiologies for this condition. The purpose of this review is to critically evaluate the recent hypotheses and their possible insights on early diagnosis, prevention and treatment. [less ▲]

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See detailDysregulation of inflammation
Bureau, Fabrice ULiege; Lekeux, Pierre ULiege

in Hoffman, A.; Robinson, N. E.; Wade, J. F. (Eds.) Proceedings of a Workshop on “Inflammatory Airway Disease: Defining the Syndrome”, Havemeyer Foundation Monograph Series No. 9 (2003)

Detailed reference viewed: 19 (2 ULiège)
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See detailDysthyroidies et dyslipidemies.
Deschampheleire, M.; Luyckx, Françoise ULiege; Scheen, André ULiege

in Revue Médicale de Liège (1999), 54(9), 746-50

While overt thyroid disturbances, characterized by symptoms and/or clinical signs with abnormal serum levels of thyroid hormones, are generally associated with perturbations in the lipid profile, the ... [more ▼]

While overt thyroid disturbances, characterized by symptoms and/or clinical signs with abnormal serum levels of thyroid hormones, are generally associated with perturbations in the lipid profile, the situation is less clear as far as subclinical thyroid disturbances, defined by isolated abnormalities of thyroid stimulating hormone (TSH) levels, are concerned. In severe hyperthyroidism, a decrease of total cholesterol, LDL cholesterol and apoprotein B concentrations is generally observed. These biological parameters are normalized when appropriate antithyroid treatment is given. In profound hypothyroidism, on the contrary, elevated levels of total and LDL cholesterol levels are observed, which decrease after hormonal replacement. In both cases, the changes in serum levels of HDL cholesterol, triglycerides and lipoprotein (a) are less systematic, both before and after treatment. Lipid abnormalities associated with subclinical thyroid disturbances remain controversial. However, two recent meta-analyses have shown higher LDL cholesterol levels in presence of subclinical hypothyroidism and a significant reduction of such lipid abnormality after administration of thyroxine. Furthermore, they demonstrated a higher prevalence of subclinical hypothyroidism in a population with hypercholesterolaemia when compared to a population with normal cholesterol levels. Finally, a significant reduction in both total and LDL cholesterol concentrations has been reported after administration of thyroxine in a small group of hypercholesterolaemic patients with basal TSH levels in the upper range of normal values. In view of the results of the literature, strategies are proposed to help the clinician in the management of patients with overt or subclinical thyroid disturbances, associated with dyslipidaemia. [less ▲]

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See detailDysthyroïdies iatrogènes
Maiga, Ibrahima ULiege; VALDES SOCIN, Hernan Gonzalo ULiege; DELWAIDE, Jean ULiege et al

in Abstract book - Annales d'Endocrinologie : 31ème Congrès de la Société Française d'Endocrinologie, Lyon 5-8 novembre 2014 (2014, October)

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See detailDystocies équines: Données descriptives et approche de la gestion des soins intensifs
Deleuze, Stefan ULiege; de la Rebière de Pouyade, Geoffroy ULiege; Sandersen, Charlotte ULiege et al

Conference (2011, December 02)

La fréquence de fourbures rencontrées après une dystocie semble faible par rapport à la littérature. L’ajout d’héparines fractionnées aux traitements habituels de la métrite du post-partum (ocytocine ... [more ▼]

La fréquence de fourbures rencontrées après une dystocie semble faible par rapport à la littérature. L’ajout d’héparines fractionnées aux traitements habituels de la métrite du post-partum (ocytocine, lavage utérins, antibiotiques et anti-inflammatoires) semble intéressant pour réduire le risque de fourbure. [less ▲]

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See detailDystonie de type 12 : un diagnostic rare et difficile.
LEROY, Patricia ULiege; Meyer, F.; Vaessen, S. et al

in Archives de pediatrie : organe officiel de la Societe francaise de pediatrie (2017), 24(7), 637-639

We report a case of dystonia 12, also called rapid-onset dystonia-parkinsonism, which occurred in a young 12-year-old boy. Type 12 dystonia is a genetic syndrome characterized by a pathogenic mutation on ... [more ▼]

We report a case of dystonia 12, also called rapid-onset dystonia-parkinsonism, which occurred in a young 12-year-old boy. Type 12 dystonia is a genetic syndrome characterized by a pathogenic mutation on ATP1A3 gene encoding the subunit alpha 3 of Na-K-ATPase protein, resulting in neuronal dysfunctions. It remains a rare syndrome with less than 100 cases described in the literature. Its atypical presentation and its rarity may lead to a wandering diagnosis, even in some cases to a conversion hysteria diagnosis. Today, unfortunately, there is no effective treatment. [less ▲]

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See detailDystonies cervicales
DELVAUX, Valérie ULiege; GONCE, Michel ULiege

Article for general public (2002)

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